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Tests in animal models have demonstrated that CoQ10 protects against doxorubicin cardiotoxicity, possibly via antioxidant activity and restoring depleted nutrient status (Combs et al 1977, Folkers et al 1978).

Recent research has identified that CoQ10 affects the expression of genes involved in human cell signalling, metabolism and transport (Groneberg et al 2005). This mechanism may account for some of the pharmacological effects observed with

CoQ10 supplementation. Additionally, CoQ10 inhibits the formation of beta-amyloid protein in vitro (Ono et al 2005).

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