It has been known for many years that sodium intake is one of the major determinants of urinary calcium excretion. It has been estimated that urinary calcium losses increase by approximately 1 mmol per 100mmol sodium intake. Experimentally sodium intake increases calcium excretion but also induces markers of bone resorption. It is hypothesized that trabecular demineralization may occur, leading to postmenopausal changes and an increased risk of vertebral fractures and cortical erosions. Further research is required in this area.
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